有氧运动消耗脂肪，阻力训练消耗糖类。这是常见的说法。
任何运动类型对脂肪消耗，有一种关键过程
Utilization of skeletal muscle triacylglycerol during postexercise recovery in h
umans
B Kiens et al. Am J Physiol. 1998 Aug.
https://journals.physiology.org/doi/full/10.1152/ajpendo.1998.275.2.E332?rfr_dat
=cr_pub++0pubmed&url_ver=Z39.88-2003&rfr_id=ori%3Arid%3Acrossref.org
Abstract
The utilization of muscle triacylglycerols was studied during and after prolonge
d bicycle ergometer exercise to exhaustion in eight healthy young men. Two days
before exercise and in the postexercise recovery period, subjects were fed a car
bohydrate-rich diet (65-70% of energy from carbohydrates). Exercise decreased mu
scle glycogen concentrations from 533 +/- 18 to 108 +/- 10 mmol/kg dry wt, where
as muscle triacylglycerol concentrations were unaffected (49 +/- 5 before vs. 49
+/- 8 mmol/kg dry wt after exercise). During the first 18 h after exercise, mus
cle glycogen concentrations were restored to 409 +/- 20 mmol/kg dry wt. In contr
ast, muscle triacylglycerol concentrations decreased (P < 0.05) to a nadir of 38
+/- 5 mmol/kg dry wt, and muscle lipoprotein lipase activity increased by 72% c
ompared with values before exercise. Pulmonary respiratory exchange ratio values
of 0.80-0.82 indicated a relatively high fractional lipid combustion despite th
e high carbohydrate intake. From 18 to 42 h of recovery, muscle glycogen synthes
is was slow and muscle triacylglycerol concentrations and lipoprotein lipase act
ivity were restored to the preexercise values. It is concluded that muscle triac
ylglycerol concentrations are not diminished during exhaustive glycogen-depletin
g exercise. However, in the postexercise recovery period, muscle glycogen resynt
hesis has high metabolic priority, resulting in postexercise lipid combustion de
spite a high carbohydrate intake. It is suggested that muscle triacylglycerols,
and probably very low density lipoprotein triacylglycerols, are important in pro
viding fuel for muscle metabolism in the postexercise recovery period.
虽然这是室内脚踏车的测试，不是典型的阻力训练运动而是有氧运动的典型。
实验过程，观察到肌细胞肝糖量从533 降至 108mmol/kg，而肌细胞脂肪量在运动前后都维
持在49mmol/kg，没变化。
之后观察到训练后18小时，肌肝糖补升到409mmol/kg。
也观察到肌脂蛋白脂解酶活性提高到72%。明显表示，恢复的能源是以消耗血液脂肪酸为主
。
无论有氧或是阻力训练，恢复时，肌纤维代谢会恢复肌肝糖。为了减少葡萄糖消耗，这时就
会增加消耗脂肪酸做为身体能量来源。
任何的训练都有效。