刚看到这一段
讲到维他命C缺乏会造成嗜睡
主要原因是维他命C关系到身体内肉碱(carnitine)的合成
顺便补充,肉碱跟身体细胞能量的产生息息相关
文章内有肉碱(carnitine)的名词解释连结
出处: 奥勒冈州立大学 微营养中心 维他命C
https://lpi.oregonstate.edu/mic/vitamins/vitamin-C
以下是
原文和翻译 我直接用google翻译翻 翻不好请去怪google 别怪我 XDDDDD
我是觉得翻得很顺 google翻译真是强大
The role of vitamin C as a cofactor is also related to its redox potential.
By maintaining enzyme-bound metals in their reduced forms, vitamin C assists
mixed-function oxidases in the synthesis of several critical biomolecules (1).
These enzymes are either monooxygenases or dioxygenases (see Table 1).
Symptoms of vitamin C deficiency, such as poor wound healing and lethargy,
likely result from the impairment of these vitamin C-dependent enzymatic
reactions leading to the insufficient synthesis of
collagen, carnitine, and catecholamines (see Deficiency). Moreover,
several dioxygenases involved in the regulation of gene expression and
the maintenance of genome integrity require vitamin C as a cofactor.
Indeed, research has recently uncovered the crucial role played by enzymes,
such as the TET dioxygenases and Jumonji domain-containing histone
demethylases, in the fate of cells and tissues (see Table 1). These enzymes
contribute to the epigenetic regulation of gene expression by catalyzing
reactions involved in the demethylation of DNA and histones.
维生素C作为辅助因子的作用也与其氧化还原电位有关。通过维持还原形式
的酶结合金属，维生素C有助于合成几种关键生物分子中的混合功能氧化酶（1）。
这些酉每是单加氧酶或双加氧酶（见表1）。
维生素C缺乏的症状，例如伤口愈合不良和嗜睡，
可能是由于这些维生素C依赖性酶促反应的损害导致胶原蛋白，肉碱和儿茶酚胺的合成不足
（见缺乏症）。此外，参与调节基因表达和维持基因组完整性的几种双加氧酶需要维生素
C作为辅助因子。实际上，研究最近揭示了酶在细胞和组织的命运中发挥的关键作用，
例如TET双加氧酶和含有组蛋白去甲基化酶的Jumonji结构域（参见表1）。这些
酶通过催化参与DNA和组蛋白去甲基化的反应而促成基因表达的表观遗传调节。